Grunert S, Ebel D, Schlack W, Thämer V Intracoronary and Intravenous Magnesium Does Not Reduce Myocardial Infarct Size in a Canine Model of Regional Ischaemia and Reperfusion Journal of Clinical and Basic Cardiology 2002; 5 (1): 23-28 PDF Summary Overview
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Figure/Graphic 5A-B: Magnesium - Linksventrikulärer Druck A: Left ventricular pressure (LVP) during intravenous infusion of magnesium potassium aspartate or placebo. B: Regional wall function in the anterior (ischaemic) and posterior (non-ischaemic) wall during intracoronary infusion of magnesium aspartate or vehicle (A-upper panel) and intravenous infusion of magnesium potassium aspartate or vehicle (B-lower panel). WThSYS = systolic wall thickening; Occl. = time period of left anterior descending coronary artery occlusion; Reperfusion = time period of left anterior descending artery reperfusion; Mg i.c. = time period when magnesium aspartate or vehicle was infused into the left anterior descending artery; Infusion i.v. = time period when magnesium potassium aspartate or vehicle was infused intravenously; values are means ± SD |
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Figure/Graphic 5A-B: Magnesium - Linksventrikulärer Druck
A: Left ventricular pressure (LVP) during intravenous infusion of magnesium potassium aspartate or placebo. B: Regional wall function in the anterior (ischaemic) and posterior (non-ischaemic) wall during intracoronary infusion of magnesium aspartate or vehicle (A-upper panel) and intravenous infusion of magnesium potassium aspartate or vehicle (B-lower panel). WThSYS = systolic wall thickening; Occl. = time period of left anterior descending coronary artery occlusion; Reperfusion = time period of left anterior descending artery reperfusion; Mg i.c. = time period when magnesium aspartate or vehicle was infused into the left anterior descending artery; Infusion i.v. = time period when magnesium potassium aspartate or vehicle was infused intravenously; values are means ± SD |