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Gasser R, Roessl U, Holzwart E, Ablasser K, Friehs I, Lewinski D, Pieske B, Mächler H, Trantiner-Yates A, Tscheliessnig KH, Mangge H, Gasser S Shift from Adult to Fetal Metabolic Phenotype During Prolonged Experimental Myocardial Ischemia: A Study on the Effect of Beta Blockers upon Gene Expression of Transmembrane Glucose Transporters Journal of Clinical and Basic Cardiology 2009; 12 (1-4): 11-17 PDF Summary Overview
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Figure/Graphic 5: Cardiac metabolism Shifting cardiac metabolism to its fetal phenotype: as an anti-anginal target (mod. from [32]) of dichloracetate augments cellular glucose metabolism. Etoximir and perhexiline inhibit cellular fatty acid metabolism and both ranolazine as well as trimetazidine slow down beta oxidation. Beta-adrenergic blockade enhances GLUT1 and GLUT4 expressions. |
Figure/Graphic 5: Cardiac metabolism
Shifting cardiac metabolism to its fetal phenotype: as an anti-anginal target (mod. from [32]) of dichloracetate augments cellular glucose metabolism. Etoximir and perhexiline inhibit cellular fatty acid metabolism and both ranolazine as well as trimetazidine slow down beta oxidation. Beta-adrenergic blockade enhances GLUT1 and GLUT4 expressions. |