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1. Borchard U
The Role of the Sympathetic Nervous System in Cardiovascular Disease
Imidazolinantagonisten
Journal of Clinical and Basic Cardiology 2001; 4 (3): 175-177
There is a strong correlation between the affinity of different I1-agonists at the I1-receptor in the ventrolateral medulla (VLM) and the oral dose needed to lower blood pressure in hypertensive patients. There is no such correlation for Ki at a2-receptors (modified according to [15]) Keywords: Antagonist,
Antagonist,
blood pressure,
Blutdruck,
Clonidin,
Diagramm,
hypertension,
Hypertonie,
Imidazolin,
Imidazoline,
Lofexidin,
Medulla,
Medulla,
Moxonidin,
Receptor,
Rezeptor,
Rilmenidin
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2. Borchard U
The Role of the Sympathetic Nervous System in Cardiovascular Disease
Moxonidin - Imidazolinrezeptor
Journal of Clinical and Basic Cardiology 2001; 4 (3): 175-177
Moxonidine selectively stimulates I1-receptors in the rostral ventrolateral medulla (RVLM). The sympathetic outflow from the RVLM to the periphery is decreased. Keywords: Imidazolin,
Imidazoline,
Medulla,
Medulla,
Moxonidin,
moxonidine,
Receptor,
Rezeptor,
Schema,
Stimulation,
Stimulation
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3. Borchard U
The Role of the Sympathetic Nervous System in Cardiovascular Disease
SNS-Überaktivität - Metabolismus
Journal of Clinical and Basic Cardiology 2001; 4 (3): 175-177
Metabolic changes due to elevated sympathetic tone: SNS overactivity leads to increased glucose production, decreased glucose utilization, increase in triglycerides and VLDL, decrease in HDL and insulin resistance (metabolic syndrome). Keywords: Cholesterin,
cholesterol,
HDL,
HDL,
insulin resistance,
Insulinresistenz,
metabolism,
Metabolismus,
overactivity,
Schema,
sympathetic nervous system,
Sympathikus,
sympathisches Nervensystem,
triglyceride,
Triglyzeride,
VLDL,
VLDL,
Überaktivität
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4. Borchard U
The Role of the Sympathetic Nervous System in Cardiovascular Disease
SNS-Überaktivität - Hypertrophie
Journal of Clinical and Basic Cardiology 2001; 4 (3): 175-177
From hypertension to heart failure: SNS overactivity leads to an increase in growth factors responsible for left ventricular hypertrophy, media hypertrophy (coronary microvessels) and fibrosis. The result may be heart failure. Keywords: Hypertrophie,
Hypertrophy,
overactivity,
Schema,
sympathetic nervous system,
Sympathikus,
sympathisches Nervensystem,
ventricle,
Ventrikel,
Überaktivität
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5. Borchard U
The Role of the Sympathetic Nervous System in Cardiovascular Disease
SNS-Überaktivität - kardiovaskuläre Erkrankung
Journal of Clinical and Basic Cardiology 2001; 4 (3): 175-177
Sustained SNS overactivity induces functional and structural changes of different organs leading to cardiovascular diseases. Keywords: cardiovascular disease,
kardiovaskuläre Erkrankung,
Schema,
sympathetic nervous system,
Sympathikus,
sympathisches Nervensystem,
Überaktivität
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6. Borchard U
The Role of the Sympathetic Nervous System in Cardiovascular Disease
Framingham - Herzfrequenz
Journal of Clinical and Basic Cardiology 2001; 4 (3): 175-177
Increase in heart rate is correlated to mortality due to coronary heart disease (CHD), cardiovascular disease or all causes (modified according to [2]) Keywords: Diagramm,
Framingham-Studie,
Framingham-Studie,
heart rate,
Herzfrequenz,
Studie,
study
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7. Borchard U
Pharmacological properties of beta-adrenoceptor blocking drugs
Betablocker - Metabolismus
Journal of Clinical and Basic Cardiology 1998; 1 (1): 5-9
Metabolic pathways of ß-adrenoceptor blocking drugs in the liver. Glu = Glucuronide. The numbers indicate the following metabolic pathways: 1) Glucuronidation of the drug at the OH group of the side chain. 2) Aromatic hydroxylation and subsequent glucuronidation partly combined with the steps described under 3 and 4 (---). 3) Oxidative desamination. 4) Ether cleavage to phenol. 5) N-desalcylation. Keywords: beta-blocker,
Betablocker,
Leber,
liver,
metabolism,
Metabolismus,
Schema
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