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7121. Mühlberger V;
Pachinger O
Koronarangiographie und PCI in Österreich im Jahr 2006 (mit Audit 2004 bis 2006)
DES - Stent - Österreich
Journal für Kardiologie - Austrian Journal of Cardiology 2008; 15 (1-2): 1-16
Anteil von Drug-eluting Stents (% DES/Stent) bezogen auf die Gesamtzahl von Stents in Prozent für alle 34 Herzkatheterlabors in Österreich 2006, aufsteigend geordnet (Mittelwerte: Österreich 69 % [= Nr. 18], Deutschland 33 % [2006],
Schweiz 82 % [2006], EU-Schnitt 26 % [2004]). (Drei Zentren haben korrigierte Daten
nachgereicht; siehe unten.) Keywords: DES,
Diagramm,
Kardiologie,
Statistik,
Österreich
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7124. Böck R;
Hoffmann S;
Podczeck-Schweighofer A
Echokardiographie aktuell: Massiv tamponierender Perikarderguss?
Echo
Journal für Kardiologie - Austrian Journal of Cardiology 2008; 15 (1-2): 42-43
Subkostaleer Farbdoppler vor thorakoskopischer Drainage Keywords: Echokardiographie,
Kardiologie
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7125. Böck R;
Hoffmann S;
Podczeck-Schweighofer A
Echokardiographie aktuell: Massiv tamponierender Perikarderguss?
Echo
Journal für Kardiologie - Austrian Journal of Cardiology 2008; 15 (1-2): 42-43
Subkostal vor thorakoskopischer Drainage Keywords: Echokardiographie,
Kardiologie
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7128. Tamse CT;
Lu X;
Mortel EG;
Cabrales E;
Feng W;
Schaefer S
The Peripheral Benzodiazepine Receptor Modulates Ca2+Transport through the VDAC in Rat Heart Mitochondria
VDAC-PBR-ANT
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 24-29
Current amplitude histograms of a VDAC-PBR-ANT
channel complex reconstituted in lipid bilayer as exposure to defined concentrations of hemin. A the holding potential of –40 mV (on trans), VDAC-PBR-ANT stayed more frequently at maximal open state. Addition of 2.5, 5 and 10 μM hemin in the cis chamber
promoted reduced current amplitudes to a lower and eventually zero value. Keywords: scheme,
VDAC-PBR-ANT
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7129. Tamse CT;
Lu X;
Mortel EG;
Cabrales E;
Feng W;
Schaefer S
The Peripheral Benzodiazepine Receptor Modulates Ca2+Transport through the VDAC in Rat Heart Mitochondria
VDAC-PBR-ANT
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 24-29
Representative current traces of VDAC-PBR-ANT channel complex in lipid bilayers. Under control conditions, VDAC-PBR-ANT isolates exhibited characteristic changes in conductance with applied
voltages (Section A). Following exposure to hemin 5–10 μM, there was gradual disappearance of conductance changes, resulting in complete channel closure after ~ 3 min (n = 7). The top trace depicts the voltage protocol used for the corresponding current. Keywords: scheme,
VDAC-PBR-ANT
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7130. Tamse CT;
Lu X;
Mortel EG;
Cabrales E;
Feng W;
Schaefer S
The Peripheral Benzodiazepine Receptor Modulates Ca2+Transport through the VDAC in Rat Heart Mitochondria
Calcium release
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 24-29
Rate of calcium release (AU/sec) from isolated mitochondria as indicated by an increase in APIII absorbance signal following calcium loading in the absence and presence of CsA under different
hemin concentrations (n = 7). There was no calcium release under control conditions in the absence of hemin. Calcium release was significantly greater than control in all experiments using hemin
2.5–10 μM with or without CsA (p < 0.01). In addition, calcium release was greater without CsA than with CsA at hemin 5 and 10 μM (*p < 0.05). Keywords: calcium,
flowchart
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7131. Tamse CT;
Lu X;
Mortel EG;
Cabrales E;
Feng W;
Schaefer S
The Peripheral Benzodiazepine Receptor Modulates Ca2+Transport through the VDAC in Rat Heart Mitochondria
Calcium Uptake - Calcium Amount
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 24-29
a) Net calcium uptake rate (AU/sec) and b) calcium amount (AU) during the initial transport of calcium into mitochondria (uptake in Figure 1) as a function of [hemin] (2.5–10 μM) in the absence and presence of CsA (n = 7). *p < 0.05 vs control. Keywords: calcium,
flowchart
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7132. Tamse CT;
Lu X;
Mortel EG;
Cabrales E;
Feng W;
Schaefer S
The Peripheral Benzodiazepine Receptor Modulates Ca2+Transport through the VDAC in Rat Heart Mitochondria
Calcium Kinetics
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 24-29
A representative tracing of extra-mitochondrial calcium kinetics following the addition of 60 μM Ca2+ into a solution containing isolated cardiac mitochondria and the calcium-sensitive marker
APIII. The absorbance (AU) increased rapidly with the addition of Ca2+ (solid arrow), followed by a decline representing uptake of calcium
by mitochondria. The slope and magnitude of this decline was quantified as calcium uptake rate and amount. Following mitochondrial loading with calcium, an increase in AU reflected calcium release
by mitochondria (dashed arrow). Calcium release was quantified as the slope of this line, and was partially inhibited by CsA. The lower trace (no hemin) shows rapid uptake of calcium and an absence of release following loading. The upper trace (hemin = 10 μM) illustrates a reduction in calcium loading as well as early release. Keywords: calcium kinetics,
flowchart
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7133. Gasser R;
Ablasser K;
Roessl U;
Kraigher-Krainer E;
Lewinski D;
Mangge H;
Mächler H;
Trantina-Yates A;
Tscheliessnigg KH;
Udermann H;
Porta S;
Friehs I;
Scherr E;
Brussee H;
Gasser S
Differential Gene Expression und Nebivolol and Atenolol during Experimental Ischemia in Human Myocardium
Nebivolol - Biological Pathway
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 16-23
Concerning nebivolol-regulated biological pathways, we could see that, during normoxia, up-regulation of pathways is hardly pronounced by nebivolol over atenolol. This becomes evident also in Figure 9a, only 3
pathways are affected here. Noteworthy here, too, the interleukin signalling pathway. Keywords: Atenolol,
flowchart,
Nebivolol
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7134. Gasser R;
Ablasser K;
Roessl U;
Kraigher-Krainer E;
Lewinski D;
Mangge H;
Mächler H;
Trantina-Yates A;
Tscheliessnigg KH;
Udermann H;
Porta S;
Friehs I;
Scherr E;
Brussee H;
Gasser S
Differential Gene Expression und Nebivolol and Atenolol during Experimental Ischemia in Human Myocardium
Pathway - Up-Regulation
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 16-23
Generally, we could see that, during experimental ischemia, upregulation of pathways is minimal by nebivolol over atenolol. This becomes
evident in Figure 9a, only 3 pathways are affected. However, the interleukin signalling pathway may be of interest in connection with inflammation, scarring
and remodelling. Keywords: Atenolol,
flowchart,
ischaemia,
Nebivolol
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7135. Gasser R;
Ablasser K;
Roessl U;
Kraigher-Krainer E;
Lewinski D;
Mangge H;
Mächler H;
Trantina-Yates A;
Tscheliessnigg KH;
Udermann H;
Porta S;
Friehs I;
Scherr E;
Brussee H;
Gasser S
Differential Gene Expression und Nebivolol and Atenolol during Experimental Ischemia in Human Myocardium
Biological Pathways
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 16-23
In well-oxygenized preparations, we can see that numerous biological pathways, mainly those involved in signalling, angiogenesis, cellular immunity, and EGF are affected by nebivolol but not by atenolol. Keywords: Atenolol,
biological pathway,
flowchart,
Nebivolol
|
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7136. Gasser R;
Ablasser K;
Roessl U;
Kraigher-Krainer E;
Lewinski D;
Mangge H;
Mächler H;
Trantina-Yates A;
Tscheliessnigg KH;
Udermann H;
Porta S;
Friehs I;
Scherr E;
Brussee H;
Gasser S
Differential Gene Expression und Nebivolol and Atenolol during Experimental Ischemia in Human Myocardium
Gene expression
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 16-23
It is interesting that nebivolol but not atenolol leads to a downregulation of gene expression in glutamine-glutamate conversion and pyruvate
metabolism, the latter of particular interest in ischemia. Keywords: Atenolol,
flowchart,
gene expression,
Nebivolol
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7137. Gasser R;
Ablasser K;
Roessl U;
Kraigher-Krainer E;
Lewinski D;
Mangge H;
Mächler H;
Trantina-Yates A;
Tscheliessnigg KH;
Udermann H;
Porta S;
Friehs I;
Scherr E;
Brussee H;
Gasser S
Differential Gene Expression und Nebivolol and Atenolol during Experimental Ischemia in Human Myocardium
Well-oxygenized preparations
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 16-23
In well-oxygenized preparations, we can see that numerous biochemical processes are up-regulated by nebivolol (not by atenolol). Interesting are reverse transcription, stress response, and protein phosphorylation. Keywords: biochemical process,
flowchart
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7138. Gasser R;
Ablasser K;
Roessl U;
Kraigher-Krainer E;
Lewinski D;
Mangge H;
Mächler H;
Trantina-Yates A;
Tscheliessnigg KH;
Udermann H;
Porta S;
Friehs I;
Scherr E;
Brussee H;
Gasser S
Differential Gene Expression und Nebivolol and Atenolol during Experimental Ischemia in Human Myocardium
Biochemical process
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 16-23
In well-oxygenized preparations, we can see that numerous biochemical processes, mainly those involved in signalling and cellular immunity, are affected by nebivolol but not by atenolol. In the lower part, we can see that many processes involved in contraction, lipid metabolism, and proliferation are down-regulated by nebivolol only (not by atenolol). This kind of
processes, if slowed down, may render the heart less vulnerable to O2 deficit or ischemia. Keywords: biochemical process,
flowchart
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7139. Gasser R;
Ablasser K;
Roessl U;
Kraigher-Krainer E;
Lewinski D;
Mangge H;
Mächler H;
Trantina-Yates A;
Tscheliessnigg KH;
Udermann H;
Porta S;
Friehs I;
Scherr E;
Brussee H;
Gasser S
Differential Gene Expression und Nebivolol and Atenolol during Experimental Ischemia in Human Myocardium
Nebivolol - Atenolol
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 16-23
Similarly, as in Figure 4, biological processes involved in cell-mediated immunity and reverse transcription are significantly up-regulated by nebivolol and not by atenolol. One may deduce that both cellular immunity
as well as reverse transcription are regulated by nebivolol at various sites. Keywords: Atenolol,
flowchart,
Nebivolol
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7140. Gasser R;
Ablasser K;
Roessl U;
Kraigher-Krainer E;
Lewinski D;
Mangge H;
Mächler H;
Trantina-Yates A;
Tscheliessnigg KH;
Udermann H;
Porta S;
Friehs I;
Scherr E;
Brussee H;
Gasser S
Differential Gene Expression und Nebivolol and Atenolol during Experimental Ischemia in Human Myocardium
Nebivolol - Atenolol
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 16-23
This figure shows biological processes more than twofold downregulated in the presence of nebivolol but unaffected by atenolol during simulated ischemia (100 % O2 replaced by 100 % N2 perfusion). One can
see that, in particular on the level of reverse transcription and T-cell-mediated immunity as well as in the process of angiogenesis, a noteworthy regulation
is brought about by nebivolol. Keywords: Atenolol,
flowchart,
Nebivolol
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|
|
7141. Gasser R;
Ablasser K;
Roessl U;
Kraigher-Krainer E;
Lewinski D;
Mangge H;
Mächler H;
Trantina-Yates A;
Tscheliessnigg KH;
Udermann H;
Porta S;
Friehs I;
Scherr E;
Brussee H;
Gasser S
Differential Gene Expression und Nebivolol and Atenolol during Experimental Ischemia in Human Myocardium
Hypoxia
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 16-23
Panels A und B show all genes regulated significantly during experimental myocardial ischemia (hypoxia) in the presence and absence of either atenolol or nebivolol. Panel A (red) summarizes all
genes down-regulated by nebivolol compared to those up-regulated by atenolol during hypoxia. N2 control indicates gene expression in ischemic preparations without betablockers. N2 nebivolol indicates gene expression in the presence of nebivolol in ischemic preparations.
The red and green lines indicate the direction of gene regulation. On the left side of panel A, N2 atenolol represents genes upregulated under atenolol during hypoxia. Panel B, accordingly, shows all genes up-regulated by nebivolol which are down-regulated
by atenolol (N2 control here also representing non-treated controls). Keywords: Atenolol,
hypoxia,
Nebivolol
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7142. Gasser R;
Ablasser K;
Roessl U;
Kraigher-Krainer E;
Lewinski D;
Mangge H;
Mächler H;
Trantina-Yates A;
Tscheliessnigg KH;
Udermann H;
Porta S;
Friehs I;
Scherr E;
Brussee H;
Gasser S
Differential Gene Expression und Nebivolol and Atenolol during Experimental Ischemia in Human Myocardium
Atenolol - Nebivolol
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 16-23
Panels A und B show all genes regulated by either atenolol or nebivolol under non-ischemic, well-oxygenated conditions. Panel
A (red) summarizes all genes down-regulated by nebivolol compared to those up-regulated by atenolol during normoxia. Hence, panel A demonstrates the direct influence of atenolol and nebivolol
on gene expression in well-oxygenated preparations. It can easily be seen that, under optimal conditions, without any sign of ischemia/hypoxia, the effect of nebivolol upon gene expression is quite different
from that of atenolol. O2 control indicates gene expression in well-oxygenated preparations without betablockers. O2 nebivolol indicates gene expression in the presence of nebivolol in non-ischemic,
well-oxygenated preparations. The red and green lines indicate the direction of gene regulation. On the left side of panel A, O2 atenolol represents genes differentially expressed under atenolol during control
conditions. Panel B, accordingly, shows all genes up-regulated by nebivolol which are down-regulated by atenolol (O2 control here also representing untreated controls). Keywords: Atenolol,
Nebivolol
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7143. Gasser R;
Ablasser K;
Roessl U;
Kraigher-Krainer E;
Lewinski D;
Mangge H;
Mächler H;
Trantina-Yates A;
Tscheliessnigg KH;
Udermann H;
Porta S;
Friehs I;
Scherr E;
Brussee H;
Gasser S
Differential Gene Expression und Nebivolol and Atenolol during Experimental Ischemia in Human Myocardium
Micriarray experiments
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 16-23
Typical results derived from microarray experiments. Light intensities on the microtiter plate reflect gene expression. Keywords: light intensities
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7144. Robertson DS
Blood Pressure and Kidney Function
Hydration - Dehydration
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 11-15
Hydration-dehydration of polyphosphoric acid. Derived
from [7, 8] Keywords: dehydration,
hydration,
scheme
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7145. Klainman E;
Starobin D;
Wishnizer R;
Yarmolovsky A;
Fink G
The Funcional Effect of Beta Blockers vs Vasodilators in Hypertension Treatment
Vo2 - VAT
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 8-10
Comparison of the main significant cardiopulmonary indices: peak VO2 and VAT, both showing significant differences between
the two groups. Keywords: flowchart,
VAT,
VO2
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7146. Elshamaa MF;
Sabry S;
Nabih M;
Elghoroury EA;
El-Saaid GS;
Ismail AAG
Alteration in Plasma Total Antioxidant Capacity, Cardiotoxic Lipid Peroxidation Product and C-Reactive Protein: A Possible Explantation for the Increased Cardiovascular Risk in Children on Hemodialysis
Serum CRP
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 2-7
Serum CRP levels in hemodialyzed patients (HD) and in
normal individuals. Columns represent mean ± SD. Keywords: chart,
serum CRP
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|
7147. Elshamaa MF;
Sabry S;
Nabih M;
Elghoroury EA;
El-Saaid GS;
Ismail AAG
Alteration in Plasma Total Antioxidant Capacity, Cardiotoxic Lipid Peroxidation Product and C-Reactive Protein: A Possible Explantation for the Increased Cardiovascular Risk in Children on Hemodialysis
TAC - MDA
Journal of Clinical and Basic Cardiology 2008; 11 (1-4): 2-7
Serum total antioxidant capacity (TAC) (a) and malondialdehyde (MDA) (b) levels in hemodialyzed patients (HD) and in normal individuals. Columns represent mean ± SD. Keywords: chart,
MDA,
TAC
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7148. Bochdansky T;
Laube W;
Böckelberger M
Muskuläre Funktionsdefizite nach Hüft- und Knietotalendoprothese
Gelenksstörungen
Journal für Mineralstoffwechsel & Muskuloskelettale Erkrankungen 2008; 15 (Sonderheft 1): 48-52
Flussdiagramm nach Gelenksstörungen mit den jeweiligen therapeutischen Ansatzmöglichkeiten. Keywords: Diagramm,
Gelenk,
Mineralstoffwechsel,
Therapie
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7149. Biedermann R
Der aktuelle Stellenwert der Sprunggelenksendoprothetik
Prothese
Journal für Mineralstoffwechsel & Muskuloskelettale Erkrankungen 2008; 15 (Sonderheft 1): 43-47
Patientin mit Panarthrose des Rückfußes auf Boden einer primär chronischen Polyarthritis präoperativ (a, b) sowie nach Implantation einer HINTEGRA-Sprunggelenksprothese
und simultaner Rückfußarthrodese (c, d) Keywords: Mineralstoffwechsel,
Prothese,
Röntgenbild,
Sprunggelenk
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7150. Chochole M;
Wlk MV
Ellbogen- und Handgelenksendoprothetik beim Rheumatiker - Richtlinien und Rehabilitation
Prothese
Journal für Mineralstoffwechsel & Muskuloskelettale Erkrankungen 2008; 15 (Sonderheft 1): 17-22
Postoperative Röntgenkontrolle nach Universal®-Handgelenksendoprothese Integra. Keywords: Handgelenk,
Mineralstoffwechsel,
Prothese,
Röntgenbild
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7151. Chochole M;
Wlk MV
Ellbogen- und Handgelenksendoprothetik beim Rheumatiker - Richtlinien und Rehabilitation
Prothese
Journal für Mineralstoffwechsel & Muskuloskelettale Erkrankungen 2008; 15 (Sonderheft 1): 17-22
OP-Situs nach Zementierung einer Discovery®-Ellbogenprothese Biomet Keywords: Ellbogen,
Foto,
Mineralstoffwechsel,
Prothese
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7155. Künzel KH;
Thöni H;
Hörmann R;
Künzel B;
Haid C
Schulterprothetik - rehabilitationsrelevante Aspekte
Schultergelenk
Journal für Mineralstoffwechsel & Muskuloskelettale Erkrankungen 2008; 15 (Sonderheft 1): 12-16
Frontalschnitt durch das Schultergelenk zeigt das "Missverhältnis" der Gelenkskörper. Keywords: Foto,
Mineralstoffwechsel,
Schultergelenk
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|
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7156. Haid C
Rehabilitation nach Gelenksersatz - biomechanische Aspekte
Drehmoment
Journal für Mineralstoffwechsel & Muskuloskelettale Erkrankungen 2008; 15 (Sonderheft 1): 6-11
Beim Einsatz der inversen Prothese vergrößert sich der Hebelarm der Muskulatur. Aus diesem Grund kann mit gleicher Muskelkraft ein größeres Drehmoment
erzeugt werden. Keywords: Hüftgelenk,
Mineralstoffwechsel,
Röntgenbild
|
|
|
7157. Haid C
Rehabilitation nach Gelenksersatz - biomechanische Aspekte
Drehmoment
Journal für Mineralstoffwechsel & Muskuloskelettale Erkrankungen 2008; 15 (Sonderheft 1): 6-11
Durch das Beugen des Oberkörpers nach vorne verlagert sich der Schwerpunkt. Bei gleicher Gewichtskraft und bei gleichem Kniewinkel verkleinert sich
der Normalabstand der Gewichtskraft. Dadurch verringert sich das Drehmoment. Keywords: Kniegelenk,
Mineralstoffwechsel,
Schema
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7158. Künzel KH;
Hörmann R;
Künzel B;
Haid C
Funktionelle Anatomie der Gelenke der oberen und unteren Extremität
Sprunggelenk
Journal für Mineralstoffwechsel & Muskuloskelettale Erkrankungen 2008; 15 (Sonderheft 1): 3-5
Übersicht über den Fuß eines juvenilen Skelettes (Alyzerinfärbung) ermöglicht den Einblick in den Bereich der Malleolengabel und zeigt Bandverbindungen
zwischen Talus und Calcaneus. Keywords: Foto,
Mineralstoffwechsel,
Sprunggelenk
|
|
|
7159. Künzel KH;
Hörmann R;
Künzel B;
Haid C
Funktionelle Anatomie der Gelenke der oberen und unteren Extremität
Kniegelenk
Journal für Mineralstoffwechsel & Muskuloskelettale Erkrankungen 2008; 15 (Sonderheft 1): 3-5
Mediale Ansicht des Kniegelenks in leichter Beugestellung. Sichtbar ist ein Teil des Musculus quadriceps femoris, das Ret. patellae med., das Ligamentum collaterale tibiale mit seinen Faserzügen und die Sehne des Musculus semimenbranosus. Keywords: Foto,
Kniegelenk,
Mineralstoffwechsel
|
|
|
7160. Künzel KH;
Hörmann R;
Künzel B;
Haid C
Funktionelle Anatomie der Gelenke der oberen und unteren Extremität
Hüftgelenk
Journal für Mineralstoffwechsel & Muskuloskelettale Erkrankungen 2008; 15 (Sonderheft 1): 3-5
Degenerativ veränderte Gelenkkörper der Art. coxae. Keywords: Foto,
Hüftgelenk,
Mineralstoffwechsel
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